Search results for "Vascular permeability"

showing 10 items of 38 documents

Diabetes and renin-angiotensin-aldosterone system: Implications for covid-19 patients with diabetes treatment management

2020

In the context of the COVID-19 continuous spreading, this paper focuses on the increased risk of diabetic patients regarding the metabolic control and the uncertainties related to SARS-CoV-2 infection. Chronic hyperglycaemia negatively affects the immune system, which triggers an increase of morbidity and mortality for viral infections. A key aspect of COVID-19 resides in the involvement of renin-angiotensin-aldosterone (RAAS) system that causes a cascade of reactions mediated by vasoactive peptides with implications in vasoconstriction, vascular permeability, oxidative stress remodelling and tissue injuries. Activation of RAAS at pulmonary level, is responsible for the local damage. Many q…

010405 organic chemistrybusiness.industry030209 endocrinology & metabolismContext (language use)Vascular permeabilityACEi ARBs COVID-19 Diabetes Of renin-angiotensin-aldosterone system RAAS SARS-CoV-2medicine.diseasemedicine.disease_causeBioinformatics01 natural sciences0104 chemical sciences03 medical and health sciences0302 clinical medicineImmune systemMetabolic control analysisDiabetes mellitusRenin–angiotensin systemmedicineGeneral Pharmacology Toxicology and Pharmaceuticsmedicine.symptombusinessVasoconstrictionOxidative stress
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IL-17A mediated endothelial breach promotes metastasis formation

2015

Abstract The role of the IL23/IL17A axis in tumor–immune interactions is a matter of controversy. Although some suggest that IL17A-producing T cells (TH17) can suppress tumor growth, others report that IL17A and IL23 accelerate tumor growth. Here, we systematically assessed the impact of IL17A-secreting lymphocytes in several murine models of tumor lung metastasis. Genetic fate mapping revealed that IL17A was secreted within lung metastases predominantly by γδ T cells, whereas TH17 cells were virtually absent. Using different tumor models, we found Il17a−/− mice to consistently develop fewer pulmonary tumor colonies. IL17A specifically increased blood vessel permeability and the expression …

0301 basic medicineGenetically modified mouseCancer ResearchPathologymedicine.medical_specialtyLung NeoplasmsEndotheliumImmunologyMelanoma ExperimentalVascular permeability610 Medicine & healthBiology10263 Institute of Experimental ImmunologyCapillary Permeability03 medical and health sciencesCarcinoma Lewis LungCell Line TumormedicineCell AdhesionAnimals1306 Cancer ResearchCell adhesionMice Knockout2403 ImmunologyLungMelanomaInterleukin-17Transendothelial and Transepithelial MigrationEndothelial Cellsmedicine.diseaseMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureCell culture570 Life sciences; biologyInterleukin 17Endothelium VascularNeoplasm Transplantation
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Expression of aquaporins 1 and 5 in a model of ventilator-induced lung injury and its relation to tidal volume

2016

New Findings What is the central question of this study? Although different studies have attempted to find factors that influence the expression of aquaporins (AQPs) in the lung in different situations, to date no research group has explored the expression of AQP1 and AQP5 jointly in rats mechanically ventilated with different tidal volumes in a model of ventilator-induced lung injury. What is the main finding? Mechanical ventilation with a high tidal volume causes lung injury and oedema, increasing lung permeability. In rats ventilated with a high tidal volume, the pulmonary expression of AQP1 decreases. We analysed the expression of aquaporins 1 and 5 and its relation with tidal volume in…

0301 basic medicineMechanical ventilationLungMembrane permeabilityRespiratory ratemedicine.medical_treatmentVascular permeabilityGeneral Medicinerespiratory systemLung injuryBiologyPulmonary edemamedicine.diseaserespiratory tract diseases03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structure030220 oncology & carcinogenesisAnesthesiamedicineTidal volumeExperimental Physiology
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Expression, Localization, and Significance of Vascular Permeability/Vascular Endothelial Growth Factor in Nasal Polyps

2005

Background The exact etiologic mechanisms leading to the formation of nasal polyps have remained largely obscure. A key phenomenon of this specific type of chronic inflammatory disease in nasal respiratory mucosa is remarkable edema. Vascular permeability/vascular endothelial growth factor (VPF/VEGF) plays an important role in inducing angiogenesis and modulating capillary permeability. Objective To study the expression and localization of VPF/VEGF as a putative key factor in nasal polyp development. Methods Specimens of nasal polyps (n = 12) were harvested during endonasal sinus surgery in patients with polypous chronic rhinosinusitis. Specimens of healthy nasal respiratory mucosa (n = 12)…

0301 basic medicinePathologymedicine.medical_specialty030102 biochemistry & molecular biologybusiness.industryVascular permeabilityChronic inflammatory diseasemedicine.diseaseVascular endothelial growth factor03 medical and health scienceschemistry.chemical_compound0302 clinical medicineOtorhinolaryngologychemistryotorhinolaryngologic diseasesMedicineNasal polyps030223 otorhinolaryngologybusinessAmerican Journal of Rhinology
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Lack of R-Ras Leads to Increased Vascular Permeability in Ischemic Retinopathy

2016

PURPOSE. The role of R-Ras in retinal angiogenesis and vascular permeability was evaluated in an oxygen-induced retinopathy (OIR) model using R-Ras knockout (KO) mice and in human diabetic neovascular membranes. METHODS. Mice deficient for R-Ras and their wild-type (WT) littermates were subjected to 75% oxygen from postnatal day 7 (P7) to P12 and then returned to room air. At P17 retinal vascularization was examined from whole mounts, and retinal vascular permeability was studied using Miles assay. Real-time RT-PCR, Western blotting, and immunohistochemistry were used to assess the expression of R-Ras in retina during development or in the OIR model. The degree of pericyte coverage and vasc…

0301 basic medicinePathologymedicine.medical_specialtyAngiogenesisIschemiaVascular permeabilityRetinaNeovascularization03 medical and health scienceschemistry.chemical_compoundmedicineRetinabusiness.industryretinal ischemiaRetinalKorva- nenä- ja kurkkutaudit silmätaudit - Otorhinolaryngology ophthalmologyDiabetic retinopathymedicine.diseaseeye diseasesdiabetic retinopathy030104 developmental biologymedicine.anatomical_structurechemistrysense organsneovascularizationmedicine.symptombusinessverkkokalvodiabeettinen retinopatiaRetinopathyInvestigative Opthalmology & Visual Science
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Diabetic macroangiopathy: Pathogenetic insights and novel therapeutic approaches with focus on high glucose-mediated vascular damage

2018

Diabetic macroangiopathy - a specific form of accelerated atherosclerosis - is characterized by intra-plaque new vessel formation due to excessive/abnormal neovasculogenesis and angiogenesis, increased vascular permeability of the capillary vessels, and tissue edema, resulting in frequent atherosclerotic plaque hemorrhage and plaque rupture. Mechanisms that may explain the premature and rapidly progressive nature of atherosclerosis in diabetes are multiple, and to a large extent still unclear. However, mechanisms related to hyperglycemia certainly play an important role. These include a dysregulated vascular regeneration. In addition, oxidative and hyperosmolar stresses, as well as the acti…

0301 basic medicineProteomicsPhysiologyAngiogenesisAquaporinMetabolomicVascular permeability030204 cardiovascular system & hematologyDiabeteBioinformaticsAquaporins03 medical and health sciences0302 clinical medicineDiabetes mellitusMedicineMetabolomicsMacrovascular diseasePharmacologybusiness.industryAquaporinRegeneration (biology)DiabetesPlaque rupturemedicine.diseaseAtherosclerosisAquaporins; Atherosclerosis; Diabetes; Hyperglycemia; Metabolomics; Proteomics030104 developmental biologyAtherosclerosiHyperglycemiaHigh glucoseMolecular Medicinebusiness
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Plasma levels of soluble vascular endothelial growth factor receptor-1 may determine the onset of early and late ovarian hyperstimulation syndrome

2006

BACKGROUND Ovarian hyperstimulation syndrome (OHSS) is a life-threatening condition associated with ovarian stimulation. Its pathophysiology is unknown and its treatment continues to be empirical. Early (E)- and late (L)-OHSS occur in women at risk, though not in all cases. Vascular endothelial growth factor (VEGF) is related to increased vascular permeability in OHSS. We analysed the dynamics of the VEGF system in E- and L-OHSS. METHODS A prospective cohort of women undergoing IVF-ICSI treatment were divided into groups. E-OHSS: Nonpregnant patients classified as women not at risk (group 1) (n = 11) and patients at risk who did not (group 2) (n = 18) and did (group 3) (n = 8) develop sever…

AdultOvulationVascular Endothelial Growth Factor Amedicine.medical_specialtyTime Factorsmedia_common.quotation_subjectOvarian hyperstimulation syndromeVascular permeabilityOvaryFertilization in VitroBiologyCohort StudiesOvarian Hyperstimulation Syndromechemistry.chemical_compoundInternal medicineBlood plasmamedicineHumansalpha-MacroglobulinsSperm Injections IntracytoplasmicProspective cohort studyOvulationmedia_commonVascular Endothelial Growth Factor Receptor-1OvaryRehabilitationObstetrics and Gynecologymedicine.diseaseVascular endothelial growth factorEndocrinologymedicine.anatomical_structureReproductive MedicinechemistryOocytesFemaleSoluble fms-like tyrosine kinase-1Human Reproduction
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Adnectin CT-322 inhibits tumor growth and affects microvascular architecture and function in Colo205 tumor xenografts

2010

Antiangiogenesis has become a promising pillar in modern cancer therapy. This study investigates the antiangiogenic effects of the PEGylated Adnectin™, CT-322, in a murine Colo-205 xenograft tumor model. CT-322 specifically binds to and blocks vascular endothelial growth factor receptor (VEGFR-2). Adnectins are a novel class of targeted biologics engineered from the 10th domain of human fibronectin. CT-322 treated tumors exhibited a significant reduction in tumor growth of 69%, a 2.8 times lower tumor surface area and fewer necrotic areas. Control tumors showed a 2.36-fold higher microvessel density (MVD) and a 2.42 times higher vessel volume in corrosion casts. The vascular architecture in…

Cancer ResearchPathologymedicine.medical_specialtyOncogenebusiness.industryCancerKinase insert domain receptorVascular permeabilityCell cyclemedicine.diseaseMolecular medicineNeovascularizationTransplantationOncologymedicinemedicine.symptombusinessInternational Journal of Oncology
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Dopamine agonist cabergoline reduces hemoconcentration and ascites in hyperstimulated women undergoing assisted reproduction.

2007

Abstract Context: Ovarian hyperstimulation syndrome (OHSS) results from increased vascular permeability (VP) caused by ovarian hypersecretion of vascular endothelial growth factor (VEGF), which activates its receptor-2. In animals, the dopamine receptor 2 agonist cabergoline (Cb2) inactivates VEGF receptor-2 and prevents increased VP. Objective: Our objective was to test whether Cb2 reduces VP and prevents OHSS in humans. Design: We conducted a prospective, randomized, double-blind study on oocyte donors at risk of developing OHSS (>20 follicles, >12 mm developed, and >20 oocytes retrieved). Interventions: Cb2 0.5 mg/d (n = 37) or a placebo (n = 32) was administered fro…

Endocrinology Diabetes and MetabolismClinical BiochemistryOvarian hyperstimulation syndromeVascular permeabilityHematocritBiochemistrychemistry.chemical_compoundHemoglobinsEndocrinologyPregnancyAscitesImage Processing Computer-AssistedMedicineProspective Studiesmedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionObstetrics and GynecologyAscitesGeneral MedicineHemoconcentrationMagnetic Resonance ImagingVascular endothelial growth factorHematocritDopamine AgonistsFemalemedicine.symptommedicine.drugAgonistAdultmedicine.medical_specialtyCabergolinemedicine.drug_classContext (language use)Fertilization in VitroDopamine agonistOvarian Hyperstimulation SyndromeDouble-Blind MethodInternal medicineCabergolineLuteal CellsHumansErgolinesGranulosa Cellsbusiness.industryReceptors Dopamine D2Peritoneal fluidBiochemistry (medical)Ovarymedicine.diseaseEndocrinologychemistryRegional Blood FlowbusinessExtracellular SpaceThe Journal of clinical endocrinology and metabolism
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Advocacy of targeting protease‐activated receptors in severe coronavirus disease 2019

2021

Identifying drug targets mitigating vascular dysfunction, thrombo-inflammation and thromboembolic complications in COVID-19 is essential. COVID-19 coagulopathy differs from sepsis coagulopathy. Factors that drive severe lung pathology and coagulation abnormalities in COVID-19 are not understood. Protein-protein interaction studies indicate that the tagged viral bait protein ORF9c directly interacts with PAR2, which modulates host cell IFN and inflammatory cytokines. In addition to direct interaction of SARS-CoV-2 viral protein with PARs, we speculate that activation of PAR by proteases plays a role in COVID-19-induced hyperinflammation. In COVID-19-associated coagulopathy elevated levels of…

InflammationPharmacologyChemokineProteasesbiologySARS-CoV-2business.industryReceptors Proteinase-ActivatedInflammationVascular permeabilitymedicine.diseaseArticleCOVID-19 Drug TreatmentProinflammatory cytokineSepsisCytokine release syndromeImmunologyCoagulopathybiology.proteinHumansMedicinemedicine.symptomCytokine Release SyndromebusinessBritish Journal of Pharmacology
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